PLOS Pathogens - Číslo 8/2012

Viewpoints

32767
Nonhuman Primate Models for HIV Cure Research

Pearls

32767
Five Mechanisms of Manipulation by Bacterial Effectors: A Ubiquitous Theme
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Trichomonosis, a Common Curable STI, and Prostate Carcinogenesis—A Proposed Molecular Mechanism
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Host Defense and Tolerance: Unique Challenges in the Placenta
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Phagocyte Responses to Protozoan Infection and How Meets the Challenge
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Fungi and the Rise of Mammals
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Bacterial Cell Surface Heterogeneity: A Pathogen's Disguise

Review

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Invariant NKT Cells: Regulation and Function during Viral Infection

Research Article

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Exon Level Transcriptomic Profiling of HIV-1-Infected CD4 T Cells Reveals Virus-Induced Genes and Host Environment Favorable for Viral Replication
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The Ebola Virus Glycoprotein Contributes to but Is Not Sufficient for Virulence
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CPAF: A Chlamydial Protease in Search of an Authentic Substrate
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Small Protease Sensitive Oligomers of PrP in Distinct Human Prions Determine Conversion Rate of PrP
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Human Monoclonal Antibody HCV1 Effectively Prevents and Treats HCV Infection in Chimpanzees
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Chemokine Receptor Ccr1 Drives Neutrophil-Mediated Kidney Immunopathology and Mortality in Invasive Candidiasis
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Telomere Length Affects the Frequency and Mechanism of Antigenic Variation in
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A Biofilm-Induced Pathway for Matrix Glucan Delivery: Implications for Drug Resistance
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The HSV-1 Exonuclease, UL12, Stimulates Recombination by a Single Strand Annealing Mechanism
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Inhibition of Fatty Acid Synthase (Fas2) Induces Mitochondrial Cell Death in Serum
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Interferon-alpha Subtype 11 Activates NK Cells and Enables Control of Retroviral Infection
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Transposon-mediated Chromosomal Integration of Transgenes in the Parasitic Nematode and Establishment of Stable Transgenic Lines
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Structural and Biochemical Basis for Development of Influenza Virus Inhibitors Targeting the PA Endonuclease
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Upregulation of Retinal Dehydrogenase 2 in Alternatively Activated Macrophages during Retinoid-dependent Type-2 Immunity to Helminth Infection in Mice
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Measles Immune Suppression: Lessons from the Macaque Model
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Cytoplasmic Entry Induces Fetal Wastage by Disrupting Maternal Foxp3 Regulatory T Cell-Sustained Fetal Tolerance

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