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Any Notes to the Pathophysiology of Secondary Cerebral Injury
Authors: P. Sklienka; I. Petrašovičová; L. Kolář; J. Jahoda; R. Hrdlička; R. Kula
Authors‘ workplace: Anesteziologicko-resuscitačná klinika FNsP Ostrava, prednosta MUDr. R. Kula, CSc.
Published in: Anest. intenziv. Med., , 2000, č. 4, s. 180-183
Category:
Overview
Authors retrospectively analysed morphologic (CT scan) and multimodal monitoring data in a group of 31 patients with cerebral injury. On theinitial CT scan cerebral edema was apparent in 79 % cases. In contrast with this observation, in course of 48 hours from injury ICP value over 20mmHg was found in 5 % and SjO2 value below 60 % was found in 59 % measurements. In SjO2 < 60 % data set significantly lower values of paCO2and significantly higher occurrence of CPP values below CPPcrit were found comparing to SjO2 > 60 % data set. Analysing causes of CPP fall belowCPPcrit 8 % participation of isolated ICP rise over ICPcrit and 67 % participation of MAP fall below MAPcrit were found. On the basis of this analysisauthors presume that ICP rise is not a cause but result of secondary cerebral injury.
Key words:
cerebral edema – intracranial pressure
Labels
Anaesthesiology, Resuscitation and Inten Intensive Care Medicine
Article was published inAnaesthesiology and Intensive Care Medicine
2000 Issue 4-
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