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Metabolic syndrome and adipose tissue: are really adipocyts responsible for everything? – editorial


Authors: M. Haluzík
Authors‘ workplace: III. interní klinika 1. lékařské fakulty UK a VFN Praha, přednosta prof. MUDr. Štěpán Svačina, DrSc., MBA
Published in: Vnitř Lék 2007; 53(1): 5-6
Category: Editorials


Sources

1. Hotamisligil GS, Shargill NS, Spiegelman BM. Adipose expression of tumor necrosis factor-alpha: direct role in obesity-linked insulin resistance. Science 1993; 259: 87-91.

2. Zhang Y, Proenca R, Maffei M et al. Positional cloning of the mouse obese gene and its human homologue. Nature 1994; 372: 425-432.

3. Xu H, Barnes GT, Yang Q et al. Chronic inflammation in fat plays a crucial role in the development of obesity-related insulin resistance. J Clin Invest 2003; 112: 1821-1830.

4. Cancello R, Henegar C, Viguerie N et al. Reduction of macrophage infiltration and chemoattractant gene expression changes in white adipose tissue of morbidly obese subjects after surgery-induced weight loss. Diabetes 2005; 54: 2277-2286.

5. Boord JB, Maeda K, Makowski L et al. Combined adipocyte-macrophage fatty acid-binding protein deficiency improves metabolism, atherosclerosis, and survival in apolipoprotein E-deficient mice. Circulation 2004; 110: 1492-1498.

6. Haluzik M, Svacina S. Metabolický syndrom a nukleární receptory PPAR. Praha: Grada Publishing 2005

Labels
Diabetology Endocrinology Internal medicine

Article was published in

Internal Medicine

Issue 1

2007 Issue 1

Most read in this issue
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