Incidence of Helicobacter Pylori in Nasal Lymphatic Tissues and its Possible Link to Adenoidal Vegetation
Authors:
R. Katra 1; Z. Kabelka 1; P. Lukeš 2; J. Astl 2; M. Jurovčík 1
Authors‘ workplace:
Klinika ušní, nosní a krční 2. LF UK a FN Motol, Praha
Subkatedra dětské otorinolaryngologie IPVZ, Praha
; přednosta doc. MUDr. Z. Kabelka
Klinika otorinolaryngologie a chirurgie hlavy a krku 1. LF UK a FN Motol, Praha
Katedra otorinolaryngologie IPVZ, Praha
1; přednosta prof. MUDr. J. Betka, DrSc.
2
Published in:
Otorinolaryngol Foniatr, 58, 2009, No. 2, pp. 102-105.
Category:
Comprehensive Reports
Overview
Helicobacter pylori (HP) is well known as the main cause of chronic gastritis, gastroduodenal ulcers and gastric cancer. It plays a role in the development of MALT lymphoma as well as other diseases. HP is also implicated in the pathogenesis of oropharyngeal . In children, the nasal lymphatic tissues and adenoids are possibly the source of this HP infection. It is believed that this infection follows a similar immune and inflammatory process to the one in the stomach or the lymphatic tissue associated to the mucosal MALT: this can lead to lymphatic hypertrophy and adenoid vegetation (AV). So far, a HP colonization has not been proved nor dismissed. Our aim is to confirm whether HP infection is linked to lymphatic hypertrophy in the nasal lymphatic tissues or it is just a concomitant occurrence.
Key words:
Helicobacter pylori (HP), adenoids, gastroesophageal reflex disease (GERD), PCR detection.
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Audiology Paediatric ENT ENT (Otorhinolaryngology)Article was published in
Otorhinolaryngology and Phoniatrics
2009 Issue 2
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